Please note that the content of this book primarily consists of articles available from Wikipedia or other free sources online. Pages: 72. Chapters: Androgen insensitivity syndrome, 5-alpha-reductase deficiency, Hermaphroditus, Ardhanarishvara, Agdistis, Intersex Society of North America, XY gonadal dysgenesis, Middlesex, Partial androgen insensitivity syndrome, Complete androgen insensitivity syndrome, Mild androgen insensitivity syndrome, The Left Hand of Darkness, Klinefelter's syndrome, Progestin-induced virilisation, Congenital adrenal hyperplasia, Lipoid congenital adrenal hyperplasia, Hermaphrodite, Sex assignment, Pseudohermaphroditism, Congenital adrenal hyperplasia due to 11 -hydroxylase deficiency, Congenital adrenal hyperplasia due to 3 beta-hydroxysteroid dehydrogenase deficiency, Herculine Barbin: Being the Recently Discovered Memoirs of a Nineteenth-century French Hermaphrodite, Trans March, The Hermaphrodite, Imposex, XX male syndrome, Transcending Boundaries Conference, 17-beta-hydroxysteroid dehydrogenase deficiency, Breast binding, Organisation Intersex International, True hermaphroditism, Mixed gonadal dysgenesis, Genital reconstructive surgery, Accord Alliance, Protogynous hermaphrodite, Androgen-induced hermaphroditism. Excerpt: Androgen insensitivity syndrome (AIS) is a condition that results in the partial or complete inability of the cell to respond to androgens. The unresponsiveness of the cell to the presence of androgenic hormones can impair or prevent the masculinization of male genitalia in the developing fetus, as well as the development of male secondary sexual characteristics at puberty, but does not significantly impair female genital or sexual development. As such, the insensitivity to androgens is only clinically significant when it occurs in genetic males (i.e. individuals with a Y-chromosome, or more specifically, an SRY gene). Clinical phenotypes in these individuals ranges from a normal male habitus with mild spermatogenic...